Abstract
Respiratory muscle dysfunction is frequently a source of morbidity and mortality in critically ill patients. Respiratory muscle dysfunction may result from heightened energetic demands placed on the inspiratory muscles due to excessive loads, reduction of inspiratory muscle strength and ventilatory reserve, or a combination of the two. If the demands are sufficiently heightened relative to the reserves, inspiratory muscle fatigue may ensue. In this review we highlight some new experimental developments primarily related to pathophysiology of respiratory musclee fatigue and weakness that may be of particular interest to the intensivist.
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