Abstract
Acetylsalicylic acid (ASA) is known to act on platelets and vessel walls. At ultra low doses it reverses the inhibitory effects produced by a vascular fragment. Use of papain on normal platelets in vitro led to the appearance of platelet aggregation without collagen induction with a range of 20.25 ± 28.91%. In the presence of vascular fragments (without ASA), this “spontaneous” aggregation remained but was reduced (13.26 ± 27.73%). This effect was reversed by ASA treatment (29.41 ± 24.17%). Reversion of vascular inhibition by ASA was not modified by papain.
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