Abstract
CORRIGENDACorrigendumPublished Online:01 Jun 2001https://doi.org/10.1152/ajprenal.2001.280.6.Fa1Original articleMoreSectionsPDF (68 KB)Download PDF ToolsExport citationAdd to favoritesGet permissionsTrack citations ShareShare onFacebookTwitterLinkedInEmailWeChat Volume 279, September 2000 Volume 48, September 2000 Pages F532–F543: Amlal H, Habo K, and Soleimani M. “Potassium deprivation upregulates expression of renal basolateral Na+-HCO3− cotransporter (NBC-1).” On p. F540, panel C of Fig. 10 was not printed. Figure 10 is reprinted below in its entirety.Fig. 10.Induction of NBC activity in mTAL cells. mTAL cells were incubated and acid loaded in a CO2/HCO3−-containing solution that lacked Na+, K+, and Cl−. A: Na+-induced pHi recovery in mTAL cells harvested from normal (Control diet) or K+-deprived rats in the presence of 2 mM amiloride and 50 nM bafilomycin A1. B: K+ deprivation induces a DIDS-sensitive dpH/dt(NBC activity) in mTAL cells of KD rats, P < 0.004.C: pHi tracing of mTAL tubules harvested from KD rats (14 days), acidified in and exposed to Na+-, K+-, and Cl−-free solution that contained 25 mM HCO3−. Each bar is mean ± SE ofn = 4–5 runs.Download figureDownload PowerPoint This article has no references to display. Download PDF Previous Back to Top Next FiguresReferencesRelatedInformationRelated ArticlesPotassium deprivation upregulates expression of renal basolateral Na+-HCO3 −cotransporter (NBC-1) 01 Sep 2000American Journal of Physiology-Renal Physiology More from this issue > Volume 280Issue 6June 2001Pages Fa1-Fa1 Copyright & PermissionsCopyright © 2001 the American Physiological Societyhttps://doi.org/10.1152/ajprenal.2001.280.6.Fa1History Published online 1 June 2001 Published in print 1 June 2001 Metrics
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
