Corrigendum

Abstract

Journal of Cellular and Molecular MedicineVolume 26, Issue 9 p. 2754-2755 CORRIGENDUMOpen Access Corrigendum This article corrects the following: Csnk1a1 inhibition modulates the inflammatory secretome and enhances response to radiotherapy in glioma Guanzheng Liu, Huan Li, Wanhong Zhang, Jiefeng Yu, Xu Zhang, Runqiu Wu, Mingshan Niu, Xuejiao Liu, Rutong Yu, Volume 25Issue 15Journal of Cellular and Molecular Medicine pages: 7395-7406 First Published online: July 3, 2021 First published: 07 May 2022 https://doi.org/10.1111/jcmm.17317AboutSectionsPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinked InRedditWechat In Guanzheng Liu et al.,1 the published article contains an error in Figure 2G. The incorrect images of EdU are used in the original publication. The corrected Figure 2 is shown below. The authors confirm that all the results and conclusions of this article remain unchanged. FIGURE 2Open in figure viewerPowerPoint Csnk1a1 downregulation and inhibition suppress GBM cell proliferation and colony formation. (A) Downregulation efficiency of Csnk1a1 silencing in LN229 and U87 cells, confirmed by immunoblotting. (B, C) Viability abilities of LN229 and U87 cells after Csnk1a1 knockdown, detected by CCK-8 assay. (D, E) Anti-proliferative effects after Csnk1a1 downregulation, determined by the EdU incorporation assay. Scale bar: 100 μm. (F) GBM cells were treated with different concentrations of D4476 for 72 h, and cell viability was determined by CCK8 assay. (G, H) The EdU incorporation assay was used to determine the anti-proliferative effect of D4476. The number of proliferating cells was normalized with the control group. Scale bar: 100 μm (*p < 0.05). (I, J) LN229 and U87 cells were treated with different concentrations of D4476 for 24 h, and the numbers of colony formed were counted, relative to the control group (*p < 0.05). (K) Csnk1a1 overexpression in LN229 and U87 cells, confirmed by immunoblotting. (L-M) Viability abilities of LN229 and U87 cells after Csnk1a1 overexpression, detected by CCK-8 assay. (N) Csnk1a1 overexpression enhances colony formation in U87 and LN229 cells. Quantitative analysis of the results of the colony formation experiment was performed (*p < 0.05). The data from three independent experiments were expressed as the means ±SEM (*p < 0.05) REFERENCE 1Liu G, Li H, Zhang W, et al. Csnk1a1 inhibition modulates the inflammatory secretome and enhances response to radiotherapy in glioma. J Cell Mol Med. 2021; 25: 7395- 7406. doi:10.1111/jcmm.16767Wiley Online LibraryCASPubMedWeb of Science®Google Scholar Volume26, Issue9May 2022Pages 2754-2755 FiguresReferencesRelatedInformation