CORRIGENDUM.

Abstract

Journal of Cellular and Molecular MedicineVolume 26, Issue 9 p. 2752-2752 CorrigendumOpen Access CORRIGENDUM This article corrects the following: MALT1 is a potential therapeutic target in glioblastoma and plays a crucial role in EGFR-induced NF-κB activation Xuejiao Liu, Chenglong Yue, Lin Shi, Guanzheng Liu, Qiyu Cao, Qianqian Shan, Yifeng Wang, Xiangyu Chen, Huan Li, Jie Wang, Shangfeng Gao, Mingshan Niu, Rutong Yu, Volume 24Issue 13Journal of Cellular and Molecular Medicine pages: 7550-7562 First Published online: May 25, 2020 First published: 07 May 2022 https://doi.org/10.1111/jcmm.17302AboutSectionsPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinked InRedditWechat In Xuejiao Liu et al.,1 the published article contains errors in Figure 5C,D. The P65 bands are incorrect in the original publication. The corrected Figure 5 is shown below. The authors confirm that all the results and conclusions of this article remain unchanged. FIGURE 5Open in figure viewerPowerPoint Knocking out MALT1 inhibits EGFR-induced NF-κB activation in GBM cells. (A and B) The effects of knocking out MALT1 on the levels of IκB-α in U87 and U251 cells as assessed by western blot analysis. (C and D) Subcellular location of p65 was detected using cellular fractionation and immunoblotting after knocking out MALT1. Lamin A/C and actin were used as nuclear and cytoplasm loading controls, respectively. (E and F) Knocking out MALT1 inhibited EGF-induced degradation of IκB-α. The cells were then stimulated with EGF (100 ng/mL) for the indicated times. IκB-α expression was analysed by western blot analysis. (G and H) MALT1 knockout suppressed EGF-induced nuclear translocation of p65 in GBM cells. The cells were stimulated with EGF (100 ng/mL) for the indicated time, and then cell lysates were subjected to western blotting using p65 antibodies. REFERENCE 1Liu X, Yue C, Shi L, et al. MALT1 is a potential therapeutic target in glioblastoma and plays a crucial role in EGFR-induced NF-κB activation. J Cell Mol Med. 2020; 24: 7550- 7562. doi:10.1111/jcmm.15383Wiley Online LibraryCASPubMedWeb of Science®Google Scholar Volume26, Issue9May 2022Pages 2752-2752 FiguresReferencesRelatedInformation