Abstract
Scientific Reports 6: Article number: 22396; published online: 03 March 2016; updated: 20 July 2016 This Article contains errors. In the last paragraph of the Results section, “Given the ability of atRA and compounds 3 and 4 to induce apoptosis in animal cancer cell line context, we surveyed severalhuman cell lines and found that CRABP1 expression was lost in human ovarian cancer cell line A2780 and human pancreatic ductal carcinoma cell line KPC”.
Highlights
In the last paragraph of the Results section, “Given the ability of atRA and compounds 3 and 4 to induce apoptosis in animal cancer cell line context, we surveyed several human cell lines and found that CRABP1 expression was lost in human ovarian cancer cell line A2780 and human pancreatic ductal carcinoma cell line KPC”
Should read: “Given the ability of atRA and compounds 3 and 4 to induce apoptosis in animal cancer cell line context, we surveyed several additional cell lines and found that CRABP1 expression was lost in human ovarian cancer cell line A2780 and mouse pancreatic ductal carcinoma cell line KPC”
To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/
Summary
“Given the ability of atRA and compounds 3 and 4 to induce apoptosis in animal cancer cell line context, we surveyed several human cell lines and found that CRABP1 expression was lost in human ovarian cancer cell line A2780 and human pancreatic ductal carcinoma cell line KPC”. OPEN Corrigendum: All trans-retinoic acid analogs promote cancer cell apoptosis through non-genomic
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