Abstract
Three hundred and fifty-four sera from a population at high risk of developing anti-IgG were examined for the presence of this antibody type and for Gm allotypic markers. Significant differences in the incidence of anti-IgG were noted between Gm phenotypes. The data suggest that the risk of anti-IgG development is related to Gm homozygosity rather that the possession of any single common Gm haplotype.
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