Abstract

HomeArteriosclerosis, Thrombosis, and Vascular BiologyVol. 37, No. 6Correction to: Extracellular Cyclophilin A, Especially Acetylated, Causes Pulmonary Hypertension by Stimulating Endothelial Apoptosis, Redox Stress, and Inflammation Free AccessCorrectionPDF/EPUBAboutView PDFView EPUBSections ToolsAdd to favoritesDownload citationsTrack citationsPermissions ShareShare onFacebookTwitterLinked InMendeleyReddit Jump toFree AccessCorrectionPDF/EPUBCorrection to: Extracellular Cyclophilin A, Especially Acetylated, Causes Pulmonary Hypertension by Stimulating Endothelial Apoptosis, Redox Stress, and Inflammation Originally published1 Jun 2017https://doi.org/10.1161/ATV.0000000000000059Arteriosclerosis, Thrombosis, and Vascular Biology. 2017;37:e68This article corrects the followingExtracellular Cyclophilin A, Especially Acetylated, Causes Pulmonary Hypertension by Stimulating Endothelial Apoptosis, Redox Stress, and InflammationIn the article by Xue et al, “Extracellular Cyclophilin A, Especially Acetylated, Causes Pulmonary Hypertension by Stimulating Endothelial Apoptosis, Redox Stress, and Inflammation,” which published online ahead of print April 27, 2017 (doi: 10.1161/ATVBAHA.117.309212), a correction was needed.Between publishing online ahead of print and final publication in the June 2017 issue, the authors have changed from an Open Access license type of CC-BY-NC-ND to a standard Copyright Transfer Agreement.The change is reflected in this issue and is available at http://atvb.ahajournals.org/content/37/6/1138. Previous Back to top Next FiguresReferencesRelatedDetailsCited By Rai N, Sydykov A, Kojonazarov B, Wilhelm J, Manaud G, Veeroju S, Ruppert C, Perros F, Ghofrani H, Weissmann N, Seeger W, Schermuly R and Novoyatleva T (2022) Targeting peptidyl-prolyl isomerase 1 in experimental pulmonary arterial hypertension, European Respiratory Journal, 10.1183/13993003.01698-2021, 60:2, (2101698), Online publication date: 1-Aug-2022. Related articlesExtracellular Cyclophilin A, Especially Acetylated, Causes Pulmonary Hypertension by Stimulating Endothelial Apoptosis, Redox Stress, and InflammationChao Xue, et al. Arteriosclerosis, Thrombosis, and Vascular Biology. 2017;37:1138-1146 June 2017Vol 37, Issue 6 Advertisement Article InformationMetrics © 2017 American Heart Association, Inc.https://doi.org/10.1161/ATV.0000000000000059PMID: 28539497 Originally publishedJune 1, 2017 PDF download Advertisement

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