Abstract
[This corrects the article DOI: 10.1371/journal.pone.0030512.].
Highlights
Following publication, the authors conducted additional experiments to determine the genetic background of Protein kinase R (PKR) regulatory domain mutant mice used in this study [1], and in a related article demonstrated that some phenotypes were not replicated when using a different PKR mutant line on a different genetic background [2]
The genetic background of the PKR regulatory domain mutant mice was not known to the authors at the time the in vivo experiments were conducted for the PLOS ONE article, and so was not reported [1]
Following the publication of this work, the authors conducted SNP genotyping analyses and determined that PKR regulatory domain mutant mice used in the experiments were on a 98.9% 129S1/SvImJ genetic background
Summary
Infection protocol (The Glas-col Inhalation Exposure instrument was different in the two experiments as the two experiments were conducted in different BSL3 facilities.) Th timing of outcome assessments The stages assayed were same in PLOS ONE and EJI experiments)
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