Correction: Antinematode Activity of Violacein and the Role of the Insulin/IGF-1 Pathway in Controlling Violacein Sensitivity in Caenorhabditis elegans.

Francesco Ballestriero,Suhelen Egan,Anahit Penesyan,David Schleheck,Malak Daim,Jadranka Nappi,Paolo Bazzicalupo,Elia Di Schiavi

doi:10.1371/journal.pone.0210026

Abstract

[This corrects the article DOI: 10.1371/journal.pone.0109201.].

Highlights

There is an error in the eleventh paragraph of the Results section
Overexpression of DAF-16 Mutation in the gene encoding for 3-phosphoinositide-dependent protein kinase Mutation in the gene encoding for the WW domain protein 1 Double mutant in the insulin-like receptor DAF-2 and in the superoxide dismutase SOD-3 Mutated in the iron/manganese superoxide dismutase SOD-3 Mutated in the putative antimicrobial lysozyme LYS-7 Mutated in the antimicrobial peptide caenopore SPP-1 a Caenorhabditis Genetics Center, the University of Minnesota. b C. elegans Gene Knockout Project http://www.celeganskoconsortium.omrf.org
Source or reference CGCa CGCa CGCa CGCa

Summary

Introduction

There is an error in the eleventh paragraph of the Results section. The correct paragraph is: In C. elegans DAF2/DAF16 controls the expression of various effector genes including those relevant for detoxification and antimicrobial activity such as the superoxidase dismutase gene sod3 and antimicrobial genes spp-1 and lys-7 [23,38]. Strain name N2 Bristol CU1546 CB1370 IU10

Results

Conclusion