Abstract
An experimental model has been produced of acute coronary failure in response to potassium chloride injection into the lateral ventricle of the cat brain. It was found that under these conditions potassium chloride activates the sympathetic nervous system, causes a spasm of the coronary vessels, an increase in the oxygen requirements of the blood, and ischemic changes in the electrocardiogram. The above disturbances end in focal necrotic and dystrophic lesions of the myocardium.
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