Abstract

The seminal studies performed by Antzelevitch and colleagues1–6 have provided the fundamental basis for the current understanding of early repolarization/J-wave syndromes. The J wave is thought to reflect a transmural repolarization gradient resulting from the early outward current and is dictated by a higher prominence of transient outward current (Ito) in epicardial tissue than in the endocardium.3,5 Early repolarization (ER) has been defined as an elevated J point, terminal slurring of the QRS complex, and possible ST-segment elevation.

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