Abstract
The incidence of congenital malformation is difficult to gauge accurately. In Philadelphia, in the late 1930's, 2.9% of 7,478 cases of still-birth had malformations recorded on the death certificates. About the same time, a rate of 20.2% was derived from autopsy reports on 435 still-births in Edinburgh. The percentage of children who died in the neonatal period with abnormalities varied between 10.5% in Edinburgh, in Glasgow and 13% in Belfast. There has been no increase in the incidence of congenital defect recently.Hydrocephalus comes second to anencephaly as a cause of death from congenital abnormality.The rarity of malformations causing hydrocephalus makes elucidation of aetiology difficult. but significant correlation has been found in some instances.
Highlights
The incidence of congenital malformation is difficult to gauge accurately
The rarity of malformations causing hydrocephalus makes elucidation of aetiology difficult. but significant correlation has been found in some instances
ISSN: 2051-7580 (Online) ISSN: 0482-3206 (Print) Res Medica is published by the Royal Medical Society, 5/5 Bristo Square, Edinburgh, EH8 9AL
Summary
Based on a Dissertation read before the Royal Medical Society on Friday, 13th October 1959. There has been no increase in the incidence of congenital defect recently. Much less clearly than with mongolism, the increased proportion of hydrocephalies born to mothers over 35 years of age has been noted. Between 1940-47 the incidence among children born to mothers in the [40-49] age groups was three times greater than among those of mothers under 30. Another series in 1947 showed that from mothers over 40 years the incidence was twice that from the under 30 group. Lack of Yit. A increases the proportion of hydrocephalic young born to rabbits and. Deficiency of Folic acid and Yit. B12 can be made to produce almost pure families with deformed aqueducts and hydrocephalus. They may represent failures of opening, delays in opening, or reclosing of a space
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