Concurrent Guillain-Barré syndrome, transverse myelitis and encephalitis post-Zika: A case report and review of the pathogenic role of multiple arboviral immunity

Abstract

We thank Yasri and Wiwanitkit for their comments on our review article of Zika virus (ZIKV) neurotropism [ [1] Mancera-Páez O. Román G.C. Pardo-Turriago R. Rodríguez Y. Anaya J.M. Concurrent Guillain-Barré syndrome, transverse myelitis and encephalitis post-Zika: a case report and review of the pathogenic role of multiple arboviral immunity. J. Neurol. Sci. 2018; 395: 47-53https://doi.org/10.1016/j.jns.2018.09.028 Abstract Full Text Full Text PDF PubMed Scopus (28) Google Scholar , [2] Mancera-Páez O. Román G.C. Pardo-Turriago R. Rodríguez Y. Anaya J.M. Erratum to ‘Concurrent Guillain-Barré syndrome, transverse myelitis and encephalitis post-Zika: a case report and review of the pathogenic role of multiple arboviral immunity’. J. Neurol. Sci. 2018; 395: 47-53https://doi.org/10.1016/j.jns.2018.10.021 Abstract Full Text Full Text PDF PubMed Scopus (1) Google Scholar ]. They indicate that in their geographic region in South East Asia (Thailand) —a ZIKV endemic area— the concurrent immunoreactivity to ZIKV and other arboviruses is very common, particularly with dengue virus (DENV) and Chikungunya virus (CHIKV) [ [3] Suwanmanee S. Surasombatpattana P. Soonthornworasiri N. Hamel R. Maneekan P. Missé D. Luplertlop N. Monitoring arbovirus in Thailand: surveillance of dengue, chikungunya and zika virus, with a focus on coinfections. Acta Trop. 2018; 188: 244-250https://doi.org/10.1016/j.actatropica.2018.09.012 Crossref PubMed Scopus (15) Google Scholar ], but there are no reports of neurological complications resulting from co-reactivity between ZIKV and other arboviruses.