Abstract
Reward processing deficits are common in current and remitted major depressive disorder (rMDD). Neurobiological substrates that implement reward-based learning (e.g., corticostriatal circuitry) are also implicated in psychomotor symptoms. Thus, psychomotor symptoms may relate to greater reward-learning deficits in rMDD. The present study sought to test whether reward-based learning is altered in rMDD with, versus without past psychomotor symptoms and controls.
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