Abstract

Purpose: There is increasing evidence that the pathophysiological mechanisms underlying posttraumatic osteoarthritis (PTOA) may differ from those of idiopathic knee OA. In addition, patients with end-stage PTOA demonstrate significantly worse post-operative outcomes following total knee arthroplasty such as inferior patient-reported outcomes and increased complications when compared to those with primary OA. Anecdotally, those with PTOA are often offered similar treatments as primary OA patients despite there potentially being different underlying mechanisms.

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