Abstract
Infertility related to endometrial factors is one of the most poorly understood aspects of reproductive biology. A recent study revealed that the interaction between the L-selectin ligand (LSL), a potential marker of implantation upregulated in receptive human endometrium, and L-selectin, the receptor located on the hatching human blastocyst, might act as the initial step in embryonic implantation at the maternal-fetal interface. We have previously demonstrated a significant positive correlation between immunohistochemical expression of LSL in surface endometrium and embryonic implantation in donor-egg recipients.
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