Abstract
To assess the cariogenic potential of almond milk, soya milk, coconut milk and bovine milk, was evaluated to check ability to enable Streptococcus mutans association formed, acid manufacturing, as well as their ability to buffer pH transforms. The baseline non stimulated whole salivary sample (2.5-5 ml) will be collected from the students in the morning at least 1 hr after breakfast. The salivary samples (pre-test and post-test) will be collected and tested for the CFUs. Salivary samples are transported in a plastic container to the microbiological laboratory. They were then tested for the number of CFUs for S. mutans using mitis salivarius bacitracin agar. Soya milk promoted much more biofilm development, whereas sugar free almond milk promoted a little. When pH test was performed, sugar free almond milk had the lowest cation exchange capacity, whereas bovine milk had the maximum cation exchange capacity.
Highlights
1.1 Background/RationaleDental decay remains among the most common infectious diseases, and tooth decay levels in preschoolers are rising [1]
The following commercially available milk will be used in the study- Bovine milk, almond milk, coconut milk and soya milk
Estimation of the pathogenic ability of commercially available bovine milk, soya milk and coconut milk on the bacterial colonies of salivary S mutans and comparing its colony to analyse the microbial growth in saliva which will determine the most caries promoting beverage
Summary
Dental decay remains among the most common infectious diseases, and tooth decay levels in preschoolers are rising [1]. Tooth decay breakdown of tooth surface caused by acidified metabolites of bacterial fermentation of sugars of plaque [2]. It is a disease caused by multiple factors, with primary causes including crucial species of bacteria of the plaque microbiota, saliva fluid & concentration, oral health, and the nature and occurrence of lactose consumption. Consistent ingestion containing sucrose-rich meal of circumstances such as saliva inadequacy may cause ecosystem transformation in the plaque microbiota [3], supposed to lead to overproduction of acidogenic and aciduric organisms, that involves S. mutans. The infectivity of S. mutans is based on its acidogenic and aciduric characteristics, that allow the microorganism to generate acid from carbohydrate fermentation and stay alive and continue to spread in an acidic conditions
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