Abstract
Commentary: Systemic, Local, and Imaging Biomarkers of Brain Injury: More Needed, and Better Use of Those Already Established?
Highlights
Marcelo de Lima Oliveira1*, Edson Bor-Seng-Shu1, Renata Faria Simm1, Tatiana Vilas Boas1 and Paulo Henrique Pires Aguiar2
Some authors have described cerebral metabolic crisis (MC) that is related to alterations in the use of glucose for energy synthesis, traumatic brain injury (TBI), and subarachnoid hemorrhage (SAH) [4,5,6,7]
Cerebral MC can occur in the absence of high lactate levels and are classified as lactate/pyruvate ratio (L/P) type 2 characterized by decreased pyruvate synthesis
Summary
Marcelo de Lima Oliveira1* , Edson Bor-Seng-Shu1 , Renata Faria Simm1 , Tatiana Vilas Boas1 and Paulo Henrique Pires Aguiar2. Cerebral metabolism plays an important role in maintaining cell functions. The brain requires glucose as the main substrate for aerobic glycolysis to sustain the high cerebral metabolic demands.
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