Abstract
Hostility is a personality trait associated with higher risk of future cardiac events in both patients with coronary artery disease (CAD) andhealthy individuals[1], but information on underlying mechanisms is scarce. Tendencies toward exaggerated physiological reactions to acute stressors predict both accelerated progression of atherosclerosis [2–6] and the likelihood of having a future cardiac event [7–9]. Therefore, testing of relationships between hostility and physiological stress reactivity may provide mechanistic insights. Hitherto, these associations have been investigated in healthy individuals but not in patients who suffered from acute coronary events. This is described in a recent meta-analysis that, in healthy individuals, higher hostility is related to increased cardiovascular stress reactivity [10]. Heightened stress reactivity of intermediate biological risk factors for coronary heartdisease(CHD)suchasinflammatoryactivitymayalsobe involved in mediation of elevated risk for acute stress
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