Abstract
Glucocerebrosidase (GBA) gene mutations are the most frequent genetic risk factor for Parkinson's disease (PD). Relationship between GBA status and increased risk for GBA-PD is still unclear. We investigated whether glucocerebrosidase activity (GCase) and α-synuclein levels in blood cells in asymptomatic subjects carrying GBA mutations (GBA carriers) are associated with a more severe prodromal PD profile.
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