Abstract
Address: 1National Reference Centre for AIDS, Microbiology Department, School of Medicine, University of Buenos Aires, Buenos Aires, Argentina, 2Laboratory of Parasitology, Microbiology Department, School of Medicine, University of Buenos Aires, Buenos Aires, Argentina and 3Endocrinology Service, Clinic Biochemistry Department, Jose de San Martin Hospital, School of Pharmacy and Biochemistry, University of Buenos Aires, Buenos Aires, Argentina * Corresponding author
Highlights
Several factors determine the risk of HIV mother-to-child transmission (MTCT), such as co-infections in placentae from HIV-1 positive mothers with other pathogens [1,2]
Simple and double infections were carried out on a placental histoculture system and on choriocarcinoma BeWo cell line
Similar results were obtained on BeWo cells
Summary
Several factors determine the risk of HIV mother-to-child transmission (MTCT), such as co-infections in placentae from HIV-1 positive mothers with other pathogens [1,2]. One of the most important endemic zoonosis in Latin America is Chagas' disease, caused by the protozoa Trypanosoma cruzi. MTCT of T. cruzi is today one of the main transmission routes in big cities [3]. The aim of the study was to determine whether T. cruzi modifies HIV infection at tissue or cellular level of the placenta
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