Abstract
There is increasing evidence that the offspring of women with gestational diabetes during pregnancy are at increased risk for the neurocognitive abnormalities. Moreover, the exact molecular mechanism by which gestational diabetes affects the developing central nervous system (CNS) remains to be defined. In the recent decades, it found that the Insulin has a crucial role in the development and function of the brain in both fetuses and adults. The researchers found that the alteration in expression/localization of insulin receptor (InsR) in the brain may be part of the cascade of events through which gestational diabetes affects the newborn’s CNS structures. Dissecting out the mechanisms responsible for gestational diabetes-related changes in the development of CNS is helping to prevent from impaired neurocognitive functions in offspring.
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