Abstract
Background and Aims : Background Vein grafts are frequently used to bypass coronary artery occlusions. Unfortunately, vein graft disease (VGD) causes impaired patency that rapidly declines due to extensive intimal hyperplasia (IH). T-cells demonstrate both pro-and anti-atherogenic effects. Therefore, we investigated T-cell involvement in a mouse-model for VGD using pharmacological targeting of the co-stimulatory factor CD137.
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