Abstract
Background and Aims : Background Vein grafts are frequently used to bypass coronary artery occlusions. Unfortunately, vein graft disease (VGD) causes impaired patency that rapidly declines due to extensive intimal hyperplasia (IH). T-cells demonstrate both pro-and anti-atherogenic effects. Therefore, we investigated T-cell involvement in a mouse-model for VGD using pharmacological targeting of the co-stimulatory factor CD137.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Disclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.