Abstract
All through the extensive studies of recent years in rickets, little has been done to investigate craniotabes. In the past, some investigators, notably Kassowitz<sup>1</sup>and later Wieland,<sup>2</sup>concerned themselves with the etiology and pathology of this clinical condition. They found it frequently associated with rickets, and the former believed that craniotabes was a manifestation of rickets and that all rickets, therefore, was congenital in origin. Wieland,<sup>3</sup>Schloss<sup>4</sup>and Finkelstein<sup>5</sup>believed that the early congenital softenings were physiologic in nature and soon disappeared, while new softenings appearing on the hard bone later were "rachitic." Czerny<sup>6</sup>states that craniotabes makes a positive diagnosis of rickets. Marfan<sup>7</sup>says that true craniotabes is of rachitic origin, but believes syphilis to be the most common cause of craniotabes. De Stefano<sup>8</sup>states that craniotabes is a manifestation either of rickets or of inherited syphilis. Thus, we see that
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