Abstract
Parathyroid pathology is usually manifested clinically as hyperparathyroidism (HPT), resulting from parathormone (PTH) excess. PTH is the chief hormone product of the parathyroid glands and can be overproduced in several complexly interrelated settings, termed primary (PHPT), secondary (SHPT), and tertiary hyperparathyroidism (THPT). PHPT can arise sporadically or from inherited syndromes that include multiple endocrine neoplasia type 1 (MEN1), multiple endocrine neoplasia type 2A (MEN2A), isolated familial hyperparathyroidism (FHPT), and familial hyperparathyroidism-jaw tumor syndrome (FHJT). SHPT is secondary to hypocalcemia, renal insufficiency, and/or severe vitamin D deficiency. THPT by definition occurs with a history of renal failure and long-standing SHPT.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
