Abstract

Some non-motor features (e.g. dementia) present more frequently in glucocerebrosidase-associated Parkinson's Disease (GBA-PD). What is the autonomic profile in GBA-PD compared to PD non-carriers (iPD)? Is there any difference between carriers of severe (GBA-SM) vs. mild (GBA-MM) mutations?

Full Text
Published version (Free)

Talk to us

Join us for a 30 min session where you can share your feedback and ask us any queries you have

Schedule a call