Abstract
Clinical and Experimental Immunomodulation 2014.
Highlights
IL-23 and TGF-β1 expression and downstream phosphorylation of STAT3
SquaiellaBaptistao et al analyzed the mechanisms accounting for the Th2 response elicited by inactivated P. acnes or its soluble polysaccharide in an ovalbumin immediate hypersensitivity model, showing that the potentiating or suppressing effects on Th2 response seem to be related to the expression of costimulatory molecules or toll-like receptors (TLRs), on antigen presenting cells
Franz et al compares the features of the experimental infection by Francisella tularensis (Ft) in wild-type and Fc gamma receptor IIB (FcγRIIB) knockout mice vaccinated with inactivated Ft, to study the role of FcγRIIB in downregulation of the immune response after the interaction of FcγRIIB with immune complexes
Summary
IL-23 and TGF-β1 expression and downstream phosphorylation of STAT3. In this same sense, C. Lenin Pavón,[1] Oscar Bottasso,[2] Hugo Besedosky,[3] Roger Loria,[4] and Marco A. The proper function of cells, receptors, and soluble mediators involved in the immune response is associated with an individual’s health.
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