Abstract
Objectives: Chloride intracellular channel-4 (CLIC4), regulated by p53 and tumor necrosis factor-α (TNF-α), is linked to an increase in transforming growth factor-β (TGF-β) and myofibroblastic differentiation in skin carcinogenesis. We analyzed CLIC4, p53, TGF-β, TNF-α, and α-smooth muscle actin (α-SMA) immunoexpression in lip carcinogenesis.
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