Abstract
The effect of a shift in temperature on lysogens for temperature-sensitive clear mutants in each of the four P1 clear cistrons is described. Only gene c1 is induced to form phage by such a shift, so it is concluded that this gene is responsible for immunity repressor production. The growth of lysogens carrying a c2, c3, or c4 mutant stops when the temperature is shifted to that at which the clear gene cannot function, indicating that these clear plaque cistrons are also active in the prophage state. Gene c3, in addition to affecting the establishment of lysogeny, appears to be essential for vegetative phage development at early and late times after infection.
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