Abstract
To the Editor: Vitiligo is an acquired, noncontagious disorder that progressively destroys melanocytes within the epidermis, hair follicles, and possibly in other organs as well. It affects 1% to 2% of the world’s population1 with an equal frequency in males and females. There is no consensus about the mechanisms involved in the dysfunction and degeneration of melanocytes in vitiligo but there are 3 major hypotheses for the pathogenesis of vitiligo: biochemical,2 neural, and autoimmune. Recent observations support the role of altered cellular immunity, autoimmunity, and a role for cytokines in the pathogenesis of vitiligo.
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