Abstract
Background and Aims: Nonalcoholic fatty liver disease (NAFLD) has been associated with cardiovascular events, mainly patients with Non- alcoholic steatohepatitis (NASH). Proprotein convertase subtilisin/kenin type 9 (PCSK9) is secreted into the plasma by the liver and regulates lipid homeostasis by promoting degradation of the LDL receptor and possibly lipogenesis, disrupting cholesterol homeostasis. The aim of the study was to examine the effect of hepatic steatosis on circulating PCSK9 levels.
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