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Abstract
In the group of diseases termed hypersensitivity pneumonitis (HP), lung lesions generally display chronic interstitial and alveolar granulomatous inflammation.1 Because of this pathology, it has been suggested that cell-mediated hypersensitivity (CMH) contributes to the pathogenesis of the group of diseases.1 In addition, other studies using blood and lung leukocytes have added impetus to this thesis.2,3 Chronic granulomatous pulmonary inflammation is best produced experimentally by the injection of participate antigens such as BCG, Micropolyspora faeni, and antigens attached to large carriers such as Sepharose 4B.
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