Abstract
Portal vein thrombosis (PVT) was first reported in 1868 by Balfour and Stewart and is a medical condition in which the lumen of the portal vein is completely or partially obstructed due to the presence of a thrombus [1]. Inherited (Factor V Leiden and Prothrombin gene mutation G201210A, Protein C, S and Anti thrombin III deficiency) and acquired thrombophilias (Lupus Anticoagulant, myeloproliferative diseases, malignancy, surgery and trauma) account for majority of the cases of PVT.
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