Abstract
Administration of allylisopropylacetamide to rats caused an increase of liver δ-aminolevulinic acid synthetase activity and porphyrin levels. Administration of nicotinic acid to allylisopropylacetamide-treated rats significantly lowered liver δ-aminolevulinic acid synthetase activity and porphyrin levels. The antiporphyric activity of nicotinic acid was probably due to a change of the redox state, altered by administration of allylisopropylacetamide.
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