Abstract
Snake bite incidences are considered a medical emergency in many parts of the world, resulting in 2.5 million envenomations and 125,000 deaths per year. Elapid snake venom is comprised of a host of enzymes and toxins that act on various targets in the prey. Venom toxins—categorized into classes including long- and short-chain α-neurotoxins, as well as non-conventional types—mainly act on nicotinic acetylcholine receptors (nAChRs), which are the molecular switches that drive nerve impulses at the neuromuscular synapse.
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