Abstract
Background and Aims : Atherosclerosis is a chronic inflammatory disease that is caused by the accumulation of LDL particles in the intima, causing the activation of immune cells and triggering an inflammatory response. LPS is a potent activator of the innate immune response and it can be transported by lipoproteins. Since humans are much more sensitive to LPS than other mammals, and very low amounts of LPS can cause inflammation and elicit an immune response, the aim of this study is to characterize the distribution of LPS and its immunogenic portion, the 3-hydroxy fatty acids (3OHFAs), among all lipoprotein types of healthy subjects.
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