Abstract
Objective: Mice with deletions in the formin2 (fmn2) gene are hypofertile due to the excessive production of immature oocytes. fmn2 -/- oocytes are unable to correctly position the meiotic spindle apparatus during Meiosis I, resulting in a retained polar body and aberrant development. To further investigate if humans have a gene similar to the mouse fmn2 gene, we have identified the FMN2 gene in the human genome.
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