Abstract
CaV1.2 is an L-type Ca2+ channel from a family of voltage dependent Ca2+ channels(VDCC)distributed mainly in cardiac and smooth muscle, endocrine cells and neurons, which produce calcium influx in response to membrane depolarization. Interaction of calmodulin (CaM) with the C-terminus (CT) of the L-type CaV1.2 channel is crucial for Ca2+-dependent inactivation (CDI). CaM also binds to the N-terminus (NT), and a CaM-formed “bridge” between CT and NT has been proposed to control CDI.
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