Abstract
Background and Aims: Human coronary vascular smooth muscle cells (hcVSMC) become foam cells by the selective uptake of cholesteryl ester (CE) from aggregated LDL (agLDL). We developed peptides that specifically inhibited agLDL formation and intracellular CE accumulation. Despite the relevance of CE-loaded hcVSMC for atherothrombosis, knowledge about their clinical implications for Coronary Artery Disease (CAD) diagnosis is limited. The objective of this work was to identify the proteins released by CE-loaded hcVSMC to identify potential new circulating CAD biomarkers.
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