Abstract
Chronic inflammation is closely related to tumorigenesis. Chronic inflammatory mediators exert pleiotropic effects in the development of cancer. On the one hand, inflammation favors carcinogenesis, malignant transformation, tumor growth, invasion, and metastatic spread; on the other hand, inflammation can stimulate immune effector mechanisms that might limit tumor growth. Inflammatory cells involved include neutrophils, eosinophils, dendritic cells, lymphocytes, NK cells, platelets, mast cells, and monocytes/macrophages.
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