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Abstract
Multiple epidemiological and prospective studies have established that high-density lipoprotein cholesterol (HDLc) is an independent risk factor for cardiovascular disease (CVD). However, pharmacological treatments focused on HDLc increase have failed to improve vascular protection, suggesting that high-density lipoprotein (HDL) functions other than cholesterol transport and metabolism could play a key role in CVD. In this regard, the study of the HDL proteome provides an improved characterization of the particle and unveils differences among individuals that could explain divergences in HDL functionality.
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