Abstract
Varicocele is present in 15 to 20% of adolescents. It induces a raise in testicular temperatures, where it may cause progressive lesions, ultimately leading to infertility. Higher testicular temperatures have been shown to lead to a decrease in gene transcription and protein translation, besides changing the proteins synthesized by Leydig and Sertoli cells. There is a protein profile that must be expressed by germ cells and by supporting Leydig and Sertoli cells in order to guarantee fertility potential.
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