Abstract
As shown in experiments on rabbits, traumatization of the lungs and pleura in conditions of open pneumothorax is accompanied by a drop of oxygen tension in the cerebral cortex, reduction of phosphorus, adenosintriphosphoric acid and creatinphosphate content in the brain with a simultaneous rise of inorganic phosphorus and lactic acid concentration, pointing to the development of acute hypoxia in the brain. At the early states of pneurnothorax the leading role in the appearance of cerebral hypoxia is played by respiratory insufficiency and the CNS exitation. Soon disturbances of cerebral circulation, and then the general vascular insufficiency, supervene.
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