Abstract
In dogs and rabbits death from asphyxia was accompanied by accelerated blood coagulation, increased tolerance of the plasma to heparin and intensified fibrinolytic activity of the blood. Twenty five minutes later these changes were enhanced and fibrinogen concentration dropped. With the death of dogs from asphyxia and trauma, thrombi were revealed in 60 min; in the first case these thrombi were subjected to a greater destruction within the 24 h. Postmortem examination of rats in 60 min demonstrated the presence of thrombi in the heart. No thrombi were present 24 h later in rats which died of asphyxia, whereas in those which died of a trauma the thrombi were retained. With the death occurring as a result of asphyxia more favorable conditions are created for thrombolysis than after death from trauma.
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