Cesarean section without sharp dissection with the placenta in low anterior position

Abstract

with the neoplasm seems indisputable, as there was some regression of virilism after its removal and the significantly elevated urinary l?-ketosteroids returned to normal levels. There was no recurrence of virilism or rise in urinary 17ketosteroids when the carcinoma recurred and metastasized, indicating that the source of the steroids was not the neoplastic cells and was presumably the pale swollen sudanophilic stromal cells. There was no cortical hyperplasia, hyperthecosis, or stromal luteinization in the opposite ovary and thus the stromal reaction in the ovary with the neoplasm must have been a direct response to the neoplastic cells. An example of embryonal carcinoma of ovary in a 3 1 -year-old woman is reported which caused masculinization and elevation of urinary 17-ketosteroid levels. There was some regression of masculinized features after removal of the neoplasm and a return of urinary 17-ketosteroid levels to normal. The endocrinopathy was attributed to the activity of lutcinized cells in the ovarian derived stroma in and at the periphery the neoplasm.