Abstract
EARLIER studies 1 on unanesthetized rats have shown that the toxic action of O 2 at high pressure (OHP) on the central nervous system has been associated with cyclic synchronous and asynchronous changes in the O 2 availability in localized regions of the brain. The nature of these regional PO 2 changes suggest they might possibly be due to or associated with regional changes in cerebral blood flow (CBF). The present experiments were carried out in an attempt to get further and more direct evidence concerning such changes in CBF in the rat. The heated thermocouple technique, a refinement of the method used in earlier blood flow studies 2 has been used for continuously recording any regional changes in blood flow from chronically implanted flow sensors. Various modifications of this method originally designed by Gibbs 3,4 to investigate the blood flow in the internal jugular vein of man were
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