Abstract
Stroke volume is a key determinate of cardiac output. It is affected by the preload, contractility and afterload of the myocardium. It is possible to gain insight into the right and left ventricular preload following the insertion of a central venous catheter and pulmonary artery catheter to measure central venous pressure and pulmonary capillary wedge pressure, respectively. Abnormal measurements can indicate a pathophysiological state and guide management.
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