Abstract

Mycobacterium tuberculosis is the bacteria that cause tuberculosis. If the infection is not treated immediately the bacterium passes via the circulatory system and spread other organs and tissues. Pathogen will travel to the meninges and causes inflammation of membranes called as tubercular meningitis. Here, the authors report a case of tuberculous meningitis a 42- years-old male patient with the chief complaints of low grade fever with chills since 1 month, headache in frontal region since 5-6 days, altered sensorium, breathing difficulty, reduced speech and left side weakness of the body since 1 day. After admitting in the ward all investigation done like MRI brain, ECG, lumbar puncture, blood tests etc. and he diagnosed as tuberculous meningitis. Patient admitted in AVBRH in ICU, investigations done, where patient was on NIV support, nasogastric tube, foleys catheter and it has been removed as patient was improving the condition and shifted in ward. Patient medical treatment in the ward was inj. C-tri 2 gm, Inj.levoflox 500 mg, inj. thimine 100 mg, Inj.Dexamethasone 10 mg, tab. Pan 40 mg, tab. Akt4 3, Inj.manitol, tab. Lorazepam. History collection, Physical examination, neurological assessment and nursing care plan were done after all treatment patient condition has been improved.

Highlights

  • After admitting in the ward all investigation done like magnetic resonance imaging (MRI) brain, ECG, lumbar puncture, blood tests etc. and he diagnosed as tuberculous meningitis

  • Extrapulmonary tuberculosis is shown by tuberculous meningitis (TBM), which is caused by the seeding of the meninges with Mycobacterium tuberculosis bacilli (MTB).MTB is first introduced into the host by droplet inhalation infecting the alveolar macrophage

  • These immune responses can lead to complications associated with tuberculous meningitis and chronic sequela seen in patients who recover from TBM

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Summary

INTRODUCTION

Extrapulmonary tuberculosis is shown by tuberculous meningitis (TBM), which is caused by the seeding of the meninges with Mycobacterium tuberculosis bacilli (MTB).MTB is first introduced into the host by droplet inhalation infecting the alveolar macrophage. The meninges are seeded by MTB and form subependymal collections called Rich foci These foci can rupture into the subarachnoid space and cause an intense inflammatory response that causes meningitis symptoms. The exudates caused by this response can encase cranial nerves and cause nerve palsies They can entrap blood vessels causing vasculitis and block cerebral spinal fluid (CSF) flow leading to hydrocephalus. These immune responses can lead to complications associated with tuberculous meningitis and chronic sequela seen in patients who recover from TBM. This activity reviews evaluation, management, and current public health preventative measures to prevent tuberculous meningitis. Chest X-ray is suggestive of active or previous pulmonary TB in approximately 50% of cases [6]

CASE REPORT
Follow-Up and Outcomes
DISCUSSION
Strength
CONCLUSION
12. Tuberculous Meningitis
Findings
Cerebrospinal Fluid Lactate in Tubercular
Full Text
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