Abstract

We report a case of a female patient who was operated at the third relapse of an atrial myxoma caused by Carney complex. The difficult operation was performed without any complications despite extensive adhesions caused by the previous operations. The further inpatient course went without complications and the patient was discharged to the consecutive treatment on the 9th postoperative day. The echocardiographic finding postoperative showed no abnormalities.

Highlights

  • We report a case of a female patient who was operated at the third relapse of an atrial myxoma caused by Carney complex

  • Case report In 1998 a 45 year old female patient presented with complete left sided hemiparesis after a cerebral embolism

  • While focusing on the reason for this clinical presentation a large mass lesion was found in the atrium via echocardiography whereby suspicion of atrial myxoma was raised

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Summary

Discussion

As with every other organ the heart can be affected by tumor and its relapses. Primary tumors of the heart are rare. Myxomas are typically sporadic and isolated, only in around five percent of all cases this disease occurs as a familial disease [1] This group of patients are younger, frequently have multifocal tumors, but, there is no gender preference. The relapse risk for sporadically occurring myxomas varies between 1 and 3% and it is increased significantly for patients with familial aggregation or Carney complex [1,3,7]. As with sporadically occurring myxomas a resection of the cardiac tumor and its relapses should be performed in patients with Carney complex. Serious surgical problems are especially peri- and epicardiac adhesions from previous operations which increase after every relapse resection This can lead to a situation where the surgical risk is higher than the risk of death due to complications of the myxoma. After the fourth operation due to the third myxoma relapse we considered platelet aggregation inhibition for our patient as a treatment to prevent complications associated with emboli as sufficient

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Conclusion
Reynen K

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